A peer-reviewed study published in ImmunoHorizons suggests COVID-19-vaccinated individuals can transmit antibodies to unvaccinated individuals “through aerosols and droplets formed as the result of normal respiration.”
In simpler terms, those who received the experimental COVID-19 shots may shed particles onto those who did not receive the shots.
Although a Pfizer document contains an entire section on the possibility of ‘vaccine shedding,’ the phenomenon has been written off as a conspiracy theory.
Pfizer made the admission in a document titled: ‘A PHASE 1/2/3, PLACEBO-CONTROLLED, RANDOMIZED, OBSERVER-BLIND, DOSE-FINDING STUDY TO EVALUATE THE SAFETY, TOLERABILITY, IMMUNOGENICITY, AND EFFICACY OF SARS-COV-2 RNA VACCINE CANDIDATES AGAINST COVID-19 IN HEALTHY INDIVIDUALS’.
The document contains a section on the potential of exposure to the study intervention due to environmental exposure, including inhalation or skin contact.
Section 8.3.5. discusses how pregnant or breastfeeding women should report exposure to the mRNA Pfizer COVID-19 jab within 24 hours of investigator awareness.
Source – Pfizer
The peer-reviewed study provided examples of this phenomenon occurring in households with COVID-19 vaccinated parents and unvaccinated children.
“The data we show from human nasal swabs provides evidence for the aerosol transfer of Abs between immune and nonimmune hosts,” scientists from the University of Colorado found.
“Respiratory transmission of viral infection is proof that oral/nasal cavity constituents can be communicated through aerosols and/or respiratory droplets. As such, it would stand to reason that Ab present within the oral/nasal environment may also be aerosolized to some degree,” the researchers wrote.
“Nasal swabs were obtained by convenience sampling both parents and their children at the Colorado Tri-County vaccine center in Aurora, CO, who were attending vaccine appointments, not limited to SARS-CoV-2,” the researchers wrote.
Read the study’s results from ImmunoHorizons:
The extended mandates for mask wearing in both social and work environments provided a unique opportunity to evaluate the possibility of aerosolized Ab expiration from vaccinated individuals. Utilizing a flow cytometry–based MMI to detect SARS-CoV-2–specific Abs (Fig. 1A, 1B) and a method previously used to elute Ab from rehydrated dried blood spots (3, 4), we identified anti-SARS-CoV-2–specific Abs eluted from surgical face masks that were worn for 1 work day by vaccinated laboratory members. Consistent with the results reported by others, we identified both IgG and IgA in saliva from vaccinated individuals (Fig. 1C, 1D). It was therefore not surprising to detect both IgG and IgA following elution from face masks (Fig. 1C, 1D).
Given these observations, we hypothesized that droplet/aerosolized Ab transfer might occur between individuals, much like droplet/aerosolized virus particles can be exchanged by the same route. To evaluate this hypothesis, we obtained nasal swabs from children living in households in which parents or family members had varying degrees of SARS-CoV-2–specific immunity, including those unvaccinated (anti-RBD–negative, anti-nucleocapsid protein [N]–negative), vaccinated (anti-RBD–positive, anti-N–negative), and COVID-19+ (anti-RBD–positive, anti-N–positive). All children evaluated were presumed to be COVID-19–negative based on being negative for anti-N Abs. Initial comparison of nasal swabs acquired from children living in vaccinated households revealed readily detectable SARS-CoV-2–specific IgG (Fig. 1E), especially when compared with the complete deficit of SARS-CoV-2–specific Ab detected in the few nasal swabs we obtained from children in nonvaccinated households. We used the variation in parents’ levels of intranasal IgG as the basis of stratification across all children’s samples. Density plots of raw and log-transformed data from 34 adult/child pairs were used to establish Ab cutoffs for high versus low parental intranasal Ab levels. Evaluation of samples in this fashion revealed that high intranasal IgG in vaccinated parents was significantly associated (p = 0.01) with a 0.38 increase in the log-transformed intranasal IgG gMFIs within a child from the same household (Fig. 1F). This significant positive relationship was observed using either parametric or nonparametric analysis, and adjustments for the correlation within household did not alter the conclusion. Although not statistically significant, a similar trend of elevated IgA was found in the same samples.
“In other words, their findings suggest aerosol transmission of antibodies can occur between COVID-19 vaccinated parents and their children—and the tendency for this transfer is directly related to the amount of nasal or oral antibodies found in those who received vaccines,” The Epoch Times wrote.
The Epoch Times corresponded with Brian Hooker, chief scientific officer at Children’s Health Defense, for further explanation.
This type of shedding is called “passive immunization,” where antibodies—primarily IgA—are actually exchanged between individuals through respiratory droplets, Brian Hooker, chief scientific officer at Children’s Health Defense, who holds a doctorate in biochemical engineering, wrote in an email to The Epoch Times. “But this would provide minimal immunity for the ‘bystanders’ based on the fact that the original mRNA vaccines provide so little protection.”
Mr. Hooker said passive immunization could elicit autoimmunity and “all sorts of reactions” in bystanders due to a similar “molecular mimicry between the COVID-19 Ig [immunoglobulin] antibodies and human proteins.”
Studies have shown that molecular mimicry between the foreign molecules and human molecules can lead to an autoimmune response causing antibodies to function incorrectly and interact against human proteins. Autoimmunity refers to an immune reaction where the body attacks its own tissues, resulting in damage or disease.
Mr. Hooker said the study suggests that if Ig antibodies can be transmitted person-to-person, there is a possibility the spike protein generated by COVID-19 vaccines could be transmitted as well.
“This could cause immunization of the bystanders as well as problems associated with spike protein toxicity to bloodstream components and other tissues,” he added.
The researchers wrote in their discussion:
The simplest interpretation of our results is that 1) aerosol transmission of Ab can occur and that 2) the propensity for this transfer is, unsurprisingly, directly related to the amount of nasal/oral Ab found within those in the population possessing immunity. We have yet to encounter an equally parsimonious interpretation, although admittedly this does not mean one does not exist. The concept of herd immunity is a central tenant of public health vaccination campaigns. Overt blockade of infection as well as a reduction in viral transmission downstream of a breakthrough infection are widely accepted conceptual mechanisms by which vaccination-induced immunity in specific individuals protects nonimmune community members. With this in mind, it stands to reason that aerosol transmission of Abs could also contribute to host protection and represent an entirely unrecognized mechanism by which passive immune protection may be communicated.
As cross-reactivity between seasonal coronavirus spike proteins and the RBD of SARS-CoV-2 has not been observed, we do not believe that our results have been unduly influenced by subjects’ prior exposure to seasonal coronavirus. Although there is some small degree of cross-reactivity between the S2 domains of SARS-CoV-2 and the OC43 seasonal strain (5, 6), this does not extend to the S1 domain or the more limited RBD. Were this to be true, it is arguable that the course of the pandemic would have been substantially altered for the better.Advertisement
Unfortunately, the difficulty in recruiting participants from unvaccinated households in conjunction with the availability of the vaccines for children under the age of 5 y rendered continued sample acquisition unsustainable. As such, we were unable to determine whether the aerosol transfer of IgA might achieve statistical significance from increased sample evaluation, nor were we able to devise any assay suitable for determining the biological relevance of the observed aerosol transfer of IgG. However, whether Ab transfer mediates host protection will be a function of exposure, and it seems reasonable to suggest, all things being equal, that any amount of Ab transfer would prove useful to the recipient host. With the documented benefits of parental vaccination in reducing the risk of infection in the unvaccinated children in the same home (7), it is tempting to speculate that aerosol-mediated Ab transfer may have possibly contributed to the reported findings. It seems likely that nasal swabs originally collected for monitoring SARS-CoV-2 transmission in this study could be repurposed for examining SARS-CoV-2–specific IgG and IgA within the vaccinated adults as well as noninfected family members, potentially providing the statistical power necessary for validating the conclusions drawn in the current study.